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FTO Is Increased in Muscle During Type 2 Diabetes, and Its Overexpression in Myotubes Alters Insulin Signaling, Enhances Lipogenesis and ROS Production, and Induces Mitochondrial Dysfunction

A. Bravard Etienne Lefai 1 E. Meugnier 1 S. Pesenti 2 Emmanuel Disse 1 J. Vouillarmet Noël Peretti 1 R. Rabasa-Lhoret M. Laville 3 Hubert Vidal Jennifer Rieusset 1 
1 CarMeN - Cardiovasculaire, métabolisme, diabétologie et nutrition
2 ISM - Institut des Sciences du Mouvement Etienne Jules Marey
3 CHLS - Centre Hospitalier Lyon Sud [CHU - HCL]
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https://hal.archives-ouvertes.fr/hal-01982013
Contributor : emmanuelle meugnier Connect in order to contact the contributor
Submitted on : Tuesday, January 15, 2019 - 1:41:09 PM
Last modification on : Monday, March 21, 2022 - 10:24:03 AM

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  • HAL Id : hal-01982013, version 1

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HCL | CNRS | UNIV-AMU | UNIV-LYON1 | INSA-LYON | INRA | CHLS | CARMEN | INSA-GROUPE | ISM-EJM | UDL | INRAE

Citation

A. Bravard, Etienne Lefai, E. Meugnier, S. Pesenti, Emmanuel Disse, et al.. FTO Is Increased in Muscle During Type 2 Diabetes, and Its Overexpression in Myotubes Alters Insulin Signaling, Enhances Lipogenesis and ROS Production, and Induces Mitochondrial Dysfunction. Diabetes, American Diabetes Association, 2010, 60 (1), pp.258-268. ⟨hal-01982013⟩

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